Dr. Geert Vanden Bossche’s warnings that a mass “vaccination” campaign in the middle of an epidemic would lead to more infectious strains of SARS-CoV-2 has come true, and now he is warning that continued “vaccination” will lead to a more deadly strain.
The virus mutated to become resistant to vaccine-induced neutralizing antibodies. Those antibodies are now non-neutralizing and actually helped Omicron become the second most contagious virus on the planet.
Normally, non-neutralizing antibodies are also good. They mark a virus for destruction so other neutralizing antibodies and killer cells can attack a virus.
But the vaccine-induced antibodies are now helping it spread. by attaching to the N-Terminal Domain (instead of the Receptor Binding Domain) in such a way that promotes attachment to the cell surface’s ACE2 receptors.
OMICRON VARIANT
- More infective
- Less deadly
NEXT POSSIBLE VARIANT
- More infective
- More deadly
But the vaccine stopped hospitalizations and death, right? Wrong, says Vanden Bossche.
It only appears the vaccine worked because Omicron inhibits the formation of syncytia between cells in the lungs, which in turn, inhibits cell-cell direct transmission. Plus the virus was already on the decline before mass vaccinations.
Each time a new copy of the virus is made, there is a chance for an error. The virus is only one mutation away from becoming more deadly. (See section “RBD core glycan addition drives neutralization escape” of this peer reviewed paper.)
The more we vaccinate, the more we suppress herd immunity and give Omicron the mutation assistance it needs, according to Vanden Bossche.
BUT WHAT ABOUT MEASLES? Measles and smallpox are stable organisms, which do not mutate like coronaviruses. We have real vaccinations for those, which last for life.
BOOSTERS: CDC overrides FDA | Children 5-11
PEER-REVIEWED SOURCE STUDIES
Structural insight into SARS-CoV-2 neutralizing antibodies and modulation of syncytia
A potential risk for mass vaccination?
SARS-CoV-2 vaccination can elicit a CD8 T-cell dominant hepatitis
Structural basis for continued antibody evasion by the SARS-CoV-2 receptor binding domain: Mutation danger if the virus “adds a sugar molecule at the location where the antibody binds to the virus.”
Omicron outcompeted Delta, does not form syncytia, thus inhibiting cell to cell transmission
Omicron variant is highly resistant against antibody-mediated neutralization
Scientists knew from SARS experiments this would happen, but did it anyway.
OPPOSING VIEW: All the vaccines prevent death and severe disease, and that’s what matters, experts say | Vanden Bossche refutes this at 42:30 mark in video above.
MORE MUTATIONS: Omicron subvariant BA.4 has arrived
